Obesogenic environment/Related Articles: Difference between revisions
Jump to navigation
Jump to search
imported>John Stephenson mNo edit summary |
No edit summary |
||
Line 33: | Line 33: | ||
{{r|satiety}} | {{r|satiety}} | ||
{{r|cholecystokinin}} | {{r|cholecystokinin}} | ||
==Articles related by keyphrases (Bot populated)== |
Latest revision as of 16:00, 27 September 2024
- See also changes related to Obesogenic environment, or pages that link to Obesogenic environment or to this page or whose text contains "Obesogenic environment".
- Poverty and obesity [r]: Relation between obesity and diet quality, dietary energy density, and energy costs. [e]
- Aminostatic hypothesis [r]: The theory of reciprocal relationship between the serum amino acid concentration and appetite. [e]
- Energostatic hypothesis [r]: The theory that appetite is regulated by the availability of energy for the brain. [e]
- Lipostatic hypothesis [r]: The theory that the hypothalamic satiety mechanisms are sensitive to the concentration of circulating metabolites. [e]
- Healthy obesity [r]: The healthy obese phenotype is characterized by favorable cardiometabolic risk factors despite excess adipose tissue. [e]
- Normal weight obesity [r]: The combination of normal body mass index (BMI) and high body fat content is associated with a high prevalence of cardiometabolic dysregulation, metabolic syndrome, and cardiovascular risk factors. [e]
- Appetite [r]: The desire to eat food, experienced as hunger, and in mammals controlled by neural circuits in the hypothalamus. [e]
- Circadian rhythms and appetite [r]: Daily variations in the regulation of food intake. [e]
- Energy balance in pregnancy and lactation [r]: Adaptations in the control of food intake and energy expenditure in different reproductive states. [e]
- Evolution of appetite regulating systems [r]: Comparisons of the mechanisms regulating food intake and energy expenditure between species. [e]
- Glucostatic theory of appetite control [r]: The theory that changes in blood glucose concentrations or arteriovenous glucose differences are detected by glucoreceptors that affect energy intake. [e]
- Melanocortins and appetite [r]: The regulation of food intake through neuropeptides related to adrenocorticotropic hormone. [e]
- Stress and appetite [r]: The interactions between the hypothalamo-pituitary-adrenal axis and the regulation of food intake. [e]
- Food reward [r]: The brain mechanisms involved in reinforcing feeding behaviour. [e]
- Gut-brain signalling [r]: The interaction between the gastrointestinal tract and the brain. [e]
- Diabesity [r]: A term referring to the intricate relationship between type 2 diabetes and obesity. [e]
- Genetics of obesity [r]: The evidence for a genetic component to obesity in humans. [e]
- Bariatric surgery [r]: The surgical removal of body fat. [e]
- Drug treatments for obesity [r]: Treatments of obesity that are based on drugs. [e]
- Exercise and body weight [r]: Correlation between physical activity and the body mass index. [e]
- Health consequences of obesity [r]: Long-term effects of obesity on health. [e]
- Adipocyte [r]: Cell that stores fat and makes it available for use as energy. [e]
- Leptin [r]: Hormone secreted by adipocytes that regulates appetite. [e]
- Ghrelin [r]: A hormone produced by P/D1 cells lining the fundus of the human stomach that stimulate appetite. [e]
- Hypothalamus [r]: Add brief definition or description
- Arcuate nucleus [r]: Add brief definition or description
- Hunger [r]: Add brief definition or description
- Satiety [r]: Add brief definition or description
- Cholecystokinin [r]: Add brief definition or description