Talk:Mitochondrion: Difference between revisions
Jump to navigation
Jump to search
imported>David E. Volk mNo edit summary |
imported>Pierre-Alain Gouanvic ("Neurotoxicity and neuroprotection": shopuldn't we expand to non-neuro?) |
||
| Line 2: | Line 2: | ||
== High priority == | == High priority == | ||
This is a high priority Google search item--don't ask me why, we don't have an article about it yet! --[[User:Larry Sanger|Larry Sanger]] 16:58, 11 October 2007 (CDT) | This is a high priority Google search item--don't ask me why, we don't have an article about it yet! --[[User:Larry Sanger|Larry Sanger]] 16:58, 11 October 2007 (CDT) | ||
== About "Neurotoxicity and neuroprotection"== | |||
I think that the processes of necrosis and apoptosis involve free radical generation catalyzed by Ca++ (calcification of the mitochondrion). If I understood well, Ca++ in mitochondria behaves like Fe++ and Cu++ out of mitochondria: it catalyzes [[Fenton reaction]]s. | |||
Perhaps we could determine whether all this is restricted to ''neuronal'' mitochondria, or if all mitochondria eventually follow the same pathological/senescent course. | |||
[[User:Pierre-Alain Gouanvic|Pierre-Alain Gouanvic]] 01:43, 2 February 2008 (CST) | |||
Revision as of 01:43, 2 February 2008
High priority
This is a high priority Google search item--don't ask me why, we don't have an article about it yet! --Larry Sanger 16:58, 11 October 2007 (CDT)
About "Neurotoxicity and neuroprotection"
I think that the processes of necrosis and apoptosis involve free radical generation catalyzed by Ca++ (calcification of the mitochondrion). If I understood well, Ca++ in mitochondria behaves like Fe++ and Cu++ out of mitochondria: it catalyzes Fenton reactions.
Perhaps we could determine whether all this is restricted to neuronal mitochondria, or if all mitochondria eventually follow the same pathological/senescent course.
Pierre-Alain Gouanvic 01:43, 2 February 2008 (CST)