Talk:Mitochondrion
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High priority
This is a high priority Google search item--don't ask me why, we don't have an article about it yet! --Larry Sanger 16:58, 11 October 2007 (CDT)
About "Neurotoxicity and neuroprotection"
I think that the processes of necrosis and apoptosis involve free radical generation catalyzed by Ca++ (calcification of the mitochondrion). If I understood well, Ca++ in mitochondria behaves like Fe++ and Cu++ out of mitochondria: it catalyzes Fenton reactions.
Perhaps we could determine whether all this is restricted to neuronal mitochondria, or if all mitochondria eventually follow the same pathological/senescent course.
Pierre-Alain Gouanvic 01:43, 2 February 2008 (CST)