Energostatic hypothesis/Bibliography

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A list of key readings about Energostatic hypothesis.
Please sort and annotate in a user-friendly manner. For formatting, consider using automated reference wikification.

Amanda Jayne Swan 16:40, 10 October 2011 (UTC) Amanda Jayne Swan 16:33, 10 October 2011 (UTC) Amanda Jayne Swan 17:01, 10 October 2011 (UTC)

1. Andersson U, Filipsson K, Abbott CR, Woods A, Smith K, Bloom SR, et al. AMP-activated protein kinase plays a role in the control of food intake. Journal of Biological Chemistry. 2004;279(13):12005-8.

2. Booth DA. POSTABSORPTIVELY INDUCED SUPPRESSION OF APPETITE AND ENERGOSTATIC CONTROL OF FEEDING. Physiology & Behavior. 1972;9(2):199-202.

Proposed that a common metabolic measure of energy, rather than a particular nutrient, controls eating and appetite.

3. Booth DA, Lovett D, Simson PC. SUBCUTANEOUS DIALYSIS IN STUDY OF EFFECTS OF NUTRIENTS ON FEEDING. Physiology & Behavior. 1970;5(10):1201-&.

4. Friedman MI. CONTROL OF ENERGY-INTAKE BY ENERGY-METABOLISM. American Journal of Clinical Nutrition. 1995;62(5):S1096-S100.

How eating behaviour is linked with energy metabolism. Changes in liver metabolism provide signals for satiety and hunger.

5. Havel PJ. Peripheral signals conveying metabolic information to the brain: Short-term and long-term regulation of food intake and energy homeostasis. Experimental Biology and Medicine. 2001;226(11):963-77.

6. Langhans W, Egli G, Scharrer E. REGULATION OF FOOD-INTAKE BY HEPATIC OXIDATIVE-METABOLISM. Brain Research Bulletin. 1985;15(4):425-8.

How metabolites and their products affected feeding behaviour and the role of vagally mediated signals from hepatic oxidation in food intake

7. Langhans W. Fatty acid oxidation in the energostatic control of eating-A new idea. Appetite. 2008;51(3):446-51.

8. Lemagnen J, Devos M. METABOLIC CORRELATES OF MEAL ONSET IN FREE FOOD INTAKE OF RATS. Physiology & Behavior. 1970;5(7):805-&.

9. Nicolaidis S, Even PC. THE ISCHYMETRIC CONTROL OF FEEDING. International Journal of Obesity. 1990;14:35-52

Suggested that the metabolic rate is metered in the brain to affect eating. Whenever locomotion-free metabolism (MF) was low, hunger was promoted. When MF levels were replenished, hunger was prevented.